Buy Inflammation And Arterial Injury Discussion

Buy Inflammation And Arterial Injury Discussion
Buy Inflammation And Arterial Injury Discussion
After coronary stenting, which of the following cell
types would not be part of the initial injury response?
(A) Platelets
(B) Lymphocytes and macrophages
(C) α-Actin positive smooth muscle cells
(D) Neutrophils
2 How does arterial injury caused by angioplasty differ
from injury due to drug-eluting stent (DES) placement?
(A) Negative remodeling may be a major factor in
DES-related healing
(B) DESs abolish the early phases of platelet
activation and inflammatory cytokine increases
(C) Polymer coating of the stent limits migration of
smooth muscle cells
(D) Both neointimal hyperplasia and negative remodeling play major roles in the long-term
healing response to balloon angioplasty
3 All of the following may contribute to arterial injury
and inflammation, except:
(A) Stent coating with selected polymers/drugs
(B) Increased stent strut thickness and specific
geometric factors
(C) Stent deployment with struts in contact with
damaged media or lipid core
(D) Stent deployment with struts in contact with
fibrous plaque
4 Panel A of the following figure is a histopathologic
section from a rabbit artery stented with polymer and
a high dose of paclitaxel. Panel B is from an artery
stented with polymer alone. How did this high dose
of paclitaxel influence the healing response?
Circulation. 2001;104:473–479.
25
Buy Inflammation And Arterial Injury Discussion
26 900 Questions: An Interventional Cardiology Board Review
(A) Decreased intimal hyperplasia in the paclitaxelpolymer coated stent
(B) Increased inflammatory reaction in the
polymer-only stent
(C) Decreased intimal hyperplasia in the polymeronly stent
(D) Persistent fibrin deposition in the polymer-only
stent
5 The following figure shows a histopathologic analysis
1 day after implantation of a coronary stent. What
does the asterisk in panel B demonstrate?
Circulation. 1999;99:44–52.
(A) Platelet-rich thrombus
(B) Lipid-rich vulnerable plaque
(C) Site of stent strut
(D) Fibrous plaque
6 Late stent thrombosis after drug-eluting stenting has
been associated with which of the following?
(A) Warfarin anticoagulation in addition to dual
antiplatelet therapy
(B) A localized, hypersensitivity (eosinophilic, giant
cell) reaction to the polymer
Proximal stent Distal stent
Th
A
B
C D EF
GHI J
K
LM NO
Luna stain (K and L) T cells (CD45Ro) B cells (CD20) Macrophages (CD68)
Circulation. 2004;109:701–705.
(C) Implantation of DESs in ST-elevated myocardial infarction (STEMI)
(D) Geographic miss during DES implantation
7 A patient develops acute chest pain and electrocardiogram (EKG) changes within 1 minute of stent
implantation. What type of arterial injury is shown
just distal to the stent?
ABC
Prestent
A
B C
Poststent
Circulation. 2002;105:2037–2042.
(A) Acute intramural hematoma
(B) Intraprocedural stent thrombosis
(C) Spontaneous rupture of distal lipid-rich plaque
(D) Acute platelet-rich thrombus due to stentmediated hypersensitivity
8 Arterial injury due to stenting induces a systemic
inflammatory response. The following figure shows
the systemic rise of which inflammatory marker after
stenting?
Inflammation and Arterial Injury 27
Base 6 h 24 h 48 h 72 h
0.19–0.290.2–0.4
0.38–1.29
1.1–2.6
0.4–1.0 *
*
* * p <0.05
0
0.5
1
1.5
2
2.5
mg/dL
Am J Cardiol. 1998;82:515–518.
(A) Serum amyloid-A
(B) C-reactive protein (CRP)
(C) Soluble CD40 ligand (sCD40L)
(D) Interleukin (IL)-6
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