Order Contrast-associate Discussion Questions

Order Contrast-associate Discussion Questions
Order Contrast-associate Discussion Questions
1. Potential strategies for reducing the risk of contrast associated nephropathy in high-risk patients include
all of the following, except:
(A) Pre- and posthydration with half-normal saline
at the time of the procedure
(B) Limiting the volume of intravascular contrast
used
(C) Maintaining urine flow rates of 150 mL per hour
with close hemodynamic monitoring and the
use of fluid loading, dopamine, and furosemide
(D) Oral N-acetylcysteine (NAC) following the
procedure
(E) Use of nonionic, iso-osmolar contrast
.2 The mechanism by which intravascular contrast is
believed to cause nephropathy is:
(A) Direct cellular toxicity to renal tubular cells
resulting in acute tubular necrosis (ATN)
(B) Hypoxic injury due to localized hemodynamic
alterations including renal vasoconstriction
(C) Neither
(D) Both
3. In a 70-kg patient with a baseline creatinine of 2.0 mg
per dL who is undergoing cardiac catheterization and
PCI, there should be attempts to limit the contrast
dose to:
(A) 70 mL
(B) 105 mL
(C) 175 mL
(D) 210 mL
Order Contrast-associate Discussion Questions
4. Patients with contrast-associated nephropathy typically have all of the following occur during their
clinical course with the exception of:
(A) An early rise in serum creatinine usually within
4 days
(B) Recovery of serum creatinine within 2 weeks
(C) Transient need for renal replacement therapy
with hemodialysis
(D) Urinalysis with active sediment showing ‘‘granular’’ muddy casts
15 Contrast-associated nephropathy can be differentiatedfrom atheroembolic renalfailure most clearly by:
(A) Absence of cutaneous findings like livedo
reticularis
(B) Absence of peripheral eosinophilia or eosinophiluria
(C) Recovery of serum creatinine within 2 weeks
(D) Presence of a normal urinalysis
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